Adenosine A<inf>2A</inf> receptor antagonism reverses the effects of dopamine receptor antagonism on instrumental output and effort-related choice in the rat: Implications for studies of psychomotor slowing

Andrew M. Farrar; Mariana Pereira; Francisco Velasco; Jörg Hockemeyer; Christa E. Müller; John D. Salamone

doi:10.1007/s00213-006-0554-5

Adenosine A<inf>2A</inf> receptor antagonism reverses the effects of dopamine receptor antagonism on instrumental output and effort-related choice in the rat: Implications for studies of psychomotor slowing

Andrew M. Farrar, Mariana Pereira, Francisco Velasco, Jörg Hockemeyer, Christa E. Müller, John D. Salamone

Research output: Contribution to journal › Article

Original language	American English
Pages (from-to)	579-586
Number of pages	8
Journal	Psychopharmacology
DOIs	https://doi.org/10.1007/s00213-006-0554-5
State	Published - 1 Apr 2007
Externally published	Yes

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10.1007/s00213-006-0554-5

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Farrar, A. M., Pereira, M., Velasco, F., Hockemeyer, J., Müller, C. E., & Salamone, J. D. (2007). Adenosine A<inf>2A</inf> receptor antagonism reverses the effects of dopamine receptor antagonism on instrumental output and effort-related choice in the rat: Implications for studies of psychomotor slowing. Psychopharmacology, 579-586. https://doi.org/10.1007/s00213-006-0554-5

Farrar, Andrew M. ; Pereira, Mariana ; Velasco, Francisco ; Hockemeyer, Jörg ; Müller, Christa E. ; Salamone, John D. / Adenosine A<inf>2A</inf> receptor antagonism reverses the effects of dopamine receptor antagonism on instrumental output and effort-related choice in the rat: Implications for studies of psychomotor slowing. In: Psychopharmacology. 2007 ; pp. 579-586.

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Adenosine A<inf>2A</inf> receptor antagonism reverses the effects of dopamine receptor antagonism on instrumental output and effort-related choice in the rat: Implications for studies of psychomotor slowing. / Farrar, Andrew M.; Pereira, Mariana; Velasco, Francisco; Hockemeyer, Jörg; Müller, Christa E.; Salamone, John D.

In: Psychopharmacology, 01.04.2007, p. 579-586.

Research output: Contribution to journal › Article

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